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Immune cell death Recent studies have shown that classical ferroptosis activators or oxidized lipids can inadvertently lead to the death of these crucial antitumor immune cells

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The most common genetic alterations in GBM involve the inactivation of tumor suppressor genes, such as p53, PTEN, and retinoblastoma (RB1), leading to uncontrolled cell proliferation, escape from apoptosis, and genomic instability [14, 16]