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61 Marta Muzios team identified IRAK-2 and MYD88 innate immune signal transduction adapter (MyD88) as mediators for IL-1R-induced NF-B signaling, and MyD88 serves as a signal transduction adapter to mediate the binding of IRAK to IL-1R, which provides possible targets for the treatment of inflammatory diseases

*Correspondence: Zhiyi He, [email protected] This article was submitted to Neurodegeneration, a section of the journal Frontiers in Neuroscience Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers