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Inflamm Bowel Dis 2005;11:178184.PubMed 85

Previous reports have claimed that L

Moreover, cell typespecific differences in regenerative capacity, metabolic state, and expression of death-regulatory factors impose additional constraints, resulting in distinct PCD outcomes across different cellular contexts

While these shifts in the GSH/GSSG balance are essential for redox signaling and the maintenance of cellular metabolism and homeostatic function, sustained disequilibrium from excess GSSG formation ultimately increases protein mixed disulfide formation

These findings indicate that the livers of PD mouse models are affected by -syn aggregation, and -syn may propagate from the brain to the liver, subsequently inducing hepatic inflammation [42]