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Description

Early studies suggest potential benefits in conditions involving impaired intestinal motility or altered gut hormone signaling, including metabolic disorders and certain gastrointestinal diseases

Cytotoxicity detection Cytotoxicity was detected by the LDH assay (Roche, 4744926001)

BDNF and TrkB Upregulation The most extensively documented mechanism of Semax in pre-clinical research is its rapid upregulation of brain-derived neurotrophic factor (BDNF) and its primary signalling receptor, tropomyosin receptor kinase B (TrkB)