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2 weeks, 3 months, or 2 years) administered via drinking water causes limited dose-dependent nonneoplastic toxic effects in B6C3F1 mice, including reduced body weight gain, liver organ weight changes, and centrilobular liver hypertrophy at high doses, with no treatment-related lesions observed in the 2-year study, indicating mice are less susceptible to dipropylene glycol toxicity than rats [1]

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Common causes include repeated injection sites, fast injection, or incorrect technique

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Thus, these results suggest that the treatment of CAFs with NAC and raloxifene prevent the cell cycle arrest induced by CAF-derived CM in A431 cells