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Despite the well-known regulatory pathways of ferroptosis, such as the GSH/GPX4 regulatory pathway, the FSP1/CoQ10 pathway, and iron metabolism and lipid metabolism, whether there are other regulatory mechanisms is unclear

I do not believe B12 causes any of what I'm asking here

It is a tripeptide built from three amino acids: glutamate, cysteine, and glycine

This was supported by the upregulation of adipogenic markers FABP4 and PPAR, with respective increases in mRNA levels of 3.0- and 2.2-fold in comparison to undifferentiated cells [1]

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