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The advantage of this drug is that it does not target the isoform B of the IR which is involved in glucose metabolism and therefore does not induce the hyperglycaemia and metabolic toxicity observed with IGF-1R targeted therapies or TKIs ( In vitro and patient-derived xenograft (PDX) models of prostate cancer have since found evidence that PTEN and the abnormally spliced ligand independent androgen receptor variant 7 (AR-V7) could have potential use as biomarkers for response to this combination of therapy

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However, the modified version (IGF-1 LR3) is 3x more potent compared to the standard form in animal models.10 More importantly, the peptide does not produce the same damaging effects as anabolic steroids and other performance-enhancing supplements.11 Previously, research has also linked the peptide to protein / RNA synthesis, cellular production, and glucose transport

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The Xc- Cystine/Glutamate Antiporter: A Mediator of Pancreatic Cancer Growth With a Role in Drug Resistance