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GHK appears to directly target myofibroblasts, converting pathological persistent collagen deposition to physiological collagen contraction through ITG-1 activation and apoptotic elimination of excess myofibroblasts (Figure 5)

Safety of the patients was maintained by discontinuing subjects that were asymptomatic with a fasting glucose over 180 mg/dL or symptomatic with a fasting glucose lower than 180 mg/dL, in order to account for hyper- or hypersensitivity reactions

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