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doi: 10.1038/s41390-023-02747-3 195 ArjomandnejadMKopecALKeelerAM

This metabolic perturbation significantly downregulates the expression of intestinal tight junction proteins, particularly ZO-1 (Mishra et al., 2023), thereby initiating a multistep pathogenic cascade: (i) compromised intestinal barrier integrity promotes bacterial translocation and systemic dissemination of deleterious metabolites

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IAV infection increases the levels of xanthine oxidase, which is an enzyme responsible for oxygen-free radical generation in mouse serum and lung tissues, thus promoting disease pathogenesis 73

Our focus on ferroptosis revealed that luseogliflozin treatment reduced TUNEL-positive cells, increased Gpx4 levels, and decreased the levels of TfR1, along with 4-HNE and MDA, markers of lipid peroxidation, indicating suppression of ferroptosis