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Miller, C., & Thompson, J

Attention is focused on the reasons preclinical promise has not translated into clinical success, emphasizing the limitations of current research models, the urgent need for reliable biomarkers of mitochondrial dysfunction, and the complexity of targeting interconnected pathological networks

Propofol ameliorates cognitive deficits following splenectomy in aged rats by inhibiting ferroptosis via the SIRT1/Nrf2/GPX4 pathway

In addition, regular blood transfusion therapy is required for thalassemia major patients to maintain adequate hemoglobin concentrations (Viprakasit and Ekwattanakit, 2018)

Consumption of GSH can promote ferroptosis by decreasing SLC7A11 and GPX4 expression in dopaminergic neurons of PD patients