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The Kraus 2014 study reported that adipocyte-specific NNMT knockdown in mice increased adipocyte energy expenditure through a mechanism interpreted as elevated methyl-donor availability for polyamine biosynthesis and for histone methylation that modulates adipocyte gene-expression programs [2]

Does BST-1, a protein homologous to CD38, play a role in organismal NAD homeostasis

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Statistics Statistical analyses were performed using GraphPad InStat Software (San Diego, CA, USA)

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