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This mitochondrial dysfunction is a result of structural and functional changes

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Specifically, it seems to limit lipid metabolism, leading to an increase in plasmatic triglycerides, fatty acids and ketone bodies (acetoacetic and -hydroxybutyric acids), and therefore aminoacids would be used to satisfy the endogenous energy needs, as there would be an impact on the availability of energy non-originated in proteins, and this would affect new tissue growth formation 3