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The advancement of disease-modifying therapies for PD hinges on continuous research aimed at deepening the mechanistic understanding of the Nrf2NLRP3 axis and refining pharmacological strategies

To verify our hypothesis, we performed sandwich ELISAs using a rabbit anti-PV1 pAb as the capture antibody, different mAbs as detection antibodies, and the international cIPV standard (NIBSC code: 12-104) as the reference