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The mechanisms include reduction of excitotoxicity and oxidative stress, enhanced neurotrophic factor expression supporting neuronal survival, angiogenesis improving cerebral blood flow, and modulation of dopamine and serotonin systems

[DOI] [PubMed] [Google Scholar] 118.Hutchins G.D., Schwaiger M., Rosenspire K.C., Krivokapich J., Schelbert H., Kuhl D.E

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Secondary endpoints included prevalence of significant fibrosis, return to drinking, heavy drinking days, and linkage to hepatology care when recommended

Nat Chem Biol 4:609616 Vita N, Landolfi G, Basle A et al (2016) Bacterial cytosolic proteins with a high capacity for Cu(I) that protect against copper toxicity