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Collectively, our results suggest that upregulation of the glutamate transporter EAAT3 is an adaptation of EBV-infected cells to maintain cellular redox homeostasis against the virus-induced oxidative stress, and that this cellular balance could be therapeutically destroyed by targeting EAAT3 to impede EBV-associated cancers

A stronger immune system B12 supports the production and function of white blood cells

Studies meeting fewer than three criteria were retained only where they represented the sole available evidence for a specific taxon, stressor, or geographic region

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Let-7 modulates the NLRP3 pathway, enhancing the inhibition of ROS production and mitochondrial DNA damage via suppression of lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1), thereby facilitating the repair of alveolar epithelial cells (Sun et al., 2019)