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Iron death, characterized by oxidative stress due to intracellular Fe2+ accumulation, leading to large amounts of ROS production, ultimately results in cell death (161) and contributes to various cardiovascular diseases including atherosclerosis, I/R injury, HF, myocardial infarction and adriamycin cardiomyopathy (162)

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Rare cases of paternal mtDNA inheritance and heteroplasmy, however, have been reported in mammals, including humans, particularly under conditions such as ooplasmic transfer or intracytoplasmic sperm injection that delay mitochondrial degradation [67]