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Furthermore, blood analysis demonstrated that the perioperative provision of high-dose selenium led to a significant increase in serum selenium and selenoprotein P (SELENOP) levels but did not translate to an adequate downstream response, as selenium-dependent plasma glutathione peroxidase (GPx3) remained unaffected [10]

Another promising approach is silencing of genes of mitochondrial amidoxime-reducing component 1 (MTARC1) [292] and 17-hydroxysteroid dehydrogenases (HSD17B13) [276]

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10.1146/annurev.micro.091208.073600 12 ChouH

Each of these afflictions is a contributor to oxidative stress