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Cigarette smoke can reprogram cells in your airways, causing pulmonary disease to hang on after smoking ends Nicotine affects mitochondrial structure and function in human airway smooth muscle cells American Journal of Physiology Lung Cellular and Molecular Physiology American Physiological Society Alleviation of cigarette smoke induced cellular senescence in BALB c mice by the Lipoxin A4 receptor agonist BML 111 is associated with mitophagy ScienceDirect Figure 10.4, Overview of immune defects caused by smoking in the lungs The Health Consequences of Smoking50 Years of Progress NCBI Bookshelf Cigarette smoke can reprogram cells in your airways, causing COPD to hang on after smoking ends

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