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However, it should be noted that the aforementioned effects are mostly derived from acute toxin-induced injury models, whereas human PD is a chronic progressive disease

This process results in the oxidation of TXN itself, characterized by the formation of disulfide bonds between the sulfhydryl groups of cysteine residues [50]

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It is the most abundant free amino acid in the CNS, acting as an excitatory neurotransmitter (Pinky et al., 2018)

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