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New technologies are allowing selective identification of subsets or sub-states of reactive astrocytes, and have identified a role for a potentially therapeutically targetable mechanism that limits XBP1-driven pathogenic astrocyte responses

It is yet to be determined whether this process contributes to the induction of oxidative stress in tumors

Nuclear translocation and transcription of antioxidant genes Upon dissociation, NRF2 translocates into the nucleus, where it binds to antioxidant response elements (AREs) located in the promoter regions of its target genes, thereby initiating their transcription

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