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This interaction results in the formation of a complex that suppresses the ubiquitination and subsequent degradation of the transcription factor BACH1

Ibrahim H, Perl A, Smith D, Lewis T, Kon Z, Goldenberg R, et al

It aids in identifying functional elements of the cellular or tissue genome and uncovering molecular components, thereby enhancing the understanding of disease mechanisms in biological processes

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In this article, the authors demonstrated that platelets express the ligand for TREM1, and showed that the binding of this molecule to neutrophil TREM1 augments LPS-induced neutrophil activation, and ultimately the innate inflammatory response