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Based on current advancements in oxidative stress research in breast cancer, we propose several innovative therapeutic approaches targeting the ROSPCD network for future development: (1) Oxidative stress-directed small-molecule drugs: Natural compounds such as curcumin can induce apoptosis by triggering mitochondrial ROS bursts and DNA damage while enhancing ferroptosis sensitivity through GPX4 inhibition

The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance

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Although PDE5I may have positive effects on improving NO and endothelial NOS and inhibiting ROCK [179], but more clinical evidence is still needed to support it