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These neurological studies are highly relevant for Long COVID and ME/CFS patients, as they confirm ALCAR's potent ability to cross the blood-brain barrier, reduce neuroinflammation, and restore the energetic capacity of the central nervous system, regardless of the initial trigger of the cognitive dysfunction

Biochemistry (John Wiley & Sons, Hoboken, NJ, 2011)

Given that insulin resistance shifts the cardiomyocyte substrate metabolism further towards FAO, the role of FAO in cardiometabolic HFpEF remains controversial 8,9

Through the precise regulation of carnitine levels, new treatments can be developed for these complex diseases, and more effective interventions can be provided for patients (Fig

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