glutathione persister cell

glutathione persister cell Drug-tolerant cancer cells are vulnerable to GPX4 inhibition Mechanism of Drug Tolerant Persister

Name: glutathione persister cell Drug-tolerant cancer cells are vulnerable to GPX4 inhibition Mechanism of Drug Tolerant Persister
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Mechanism of Drug Tolerant Persister Cancer Cells: The Landscape and Clinical Implication for Therapy Journal of Thoracic Oncology Persister cells have a disabled antioxidant program and depend on GPX4 Download Scientific Diagram Vaccinia related kinase 2 inhibition elicits vulnerability of glutathione metabolism in pancreatic cancer Cell Death & Disease Glutathione accelerates the cell cycle and cellular reprogramming in plant regeneration: Developmental Cell Low GPX4 drives a sustained drug tolerant persister state in TNBC by a targetable adaptive FSP1 upregulation ScienceDirect

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Jin X, Li H, Li B, Zhang C, He Y

Samples were de-proteinized with 5% (w/v) sulphosalycilic acid (SSA, Sigma-Aldrich, USA) and the glutathione content was determined after dilution of the acid soluble fraction in Na-phosphate buffer containing EDTA (pH 7.5)

Conclusion ROS are the natural consequence of aerobic metabolism and are important for basic cellular functions, including proliferation, inflammation, apoptosis, and gene expression

Mancarella D, Plass C

Below, we summarize novel therapeutic targets and strategies with clinical application potential to provide more options for disease treatment (Table 1)